PIMA525333

RASD2 Monoclonal Antibody (OTI1F7), Invitrogen™

Manufacturer: Thermo Scientific

Select a Size

Pack Size SKU Availability Price
Each of 1 PIMA525333-Each-of-1 In Stock ₹ 52,287.50

PIMA525333 - Each of 1

₹ 52,287.50

In Stock

Quantity

1

Base Price: ₹ 52,287.50

GST (18%): ₹ 9,411.75

Total Price: ₹ 61,699.25

Antigen

RASD2

Classification

Monoclonal

Concentration

1 mg/mL

Formulation

PBS with 1% BSA, 50% glycerol and 0.02% sodium azide; pH 7.3

Gene Accession No.

Q96D21

Gene Symbols

RASD2

Immunogen

Full length human recombinant protein of RASD2 produced in HEK293T cell

Quantity

100μL

Primary or Secondary

Primary

Target Species

Human

Product Type

Antibody

Isotype

IgG1

Applications

Flow Cytometry, Immunocytochemistry, Immunohistochemistry (Paraffin), Western Blot

Clone

OTI1F7

Conjugate

Unconjugated

Gene

RASD2

Gene Alias

4930526B11Rik; AU045414; GTP-binding protein Rhes; MGC:4834; Ras homolog enriched in striatum; RASD family member 2; RASD family, member 2; Rasd2; Rhes; SE6C; TEM2; TEM-2; Tumor endothelial marker 2

Host Species

Mouse

Purification Method

Affinity Chromatography

Regulatory Status

RUO

Gene ID (Entrez)

23551

Content And Storage

-20° C, Avoid Freeze/Thaw Cycles

Form

Liquid

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Description

  • Rhes, also known as tumor endothelial marker 2 (TEM2), is a small GTP-binding protein that is predominantly expressed in the striatal region of the brain
  • This protein belongs to the RASD subfamily of Ras-related GTP-binding protein superfamily and is thought to play a role in the normal development and function of the brain as mice lacking this gene showed increased anxiety levels and motor coordination deficits
  • Rhes was identified as TEM2 through analysis of genes whose expression was upregulated in tumor endothelium
  • Tumor endothelial markers are significantly up-regulated during angiogenesis and neoangiogenesis that are crucial for the growth of solid tumors
  • TEMs localized on the cell surface and conserved across species are of particular interest for future development of anti-angiogenic therapies.