NeuroD1 Polyclonal Antibody, Invitrogen™
Manufacturer: Thermo Scientific
Select a Size
| Pack Size | SKU | Availability | Price |
|---|---|---|---|
| Each of 1 | PIPA582075-Each-of-1 | In Stock | ₹ 49,706.50 |
PIPA582075 - Each of 1
In Stock
Quantity
1
Base Price: ₹ 49,706.50
GST (18%): ₹ 8,947.17
Total Price: ₹ 58,653.67
Antigen
NeuroD1
Classification
Polyclonal
Conjugate
Unconjugated
Gene
NEUROD1
Gene Alias
Basic helix-loop-helix factor 1; basic helix-loop-helix transcription factor; Beta2; beta-cell E-box transactivator 2; beta-cell E-box transcriptional activator 2; BHF1; BHF-1; bHLHa3; Class A basic helix-loop-helix protein 3; MODY6; neuro d1; Neurod; Neurod1; neurogenic differentiation 1; neurogenic differentiation factor 1; neurogenic helix-loop-helix protein NEUROD; neuronal differentiation 1
Host Species
Rabbit
Purification Method
Anitgen affinity chromatography, Antigen affinity chromatography
Regulatory Status
RUO
Gene ID (Entrez)
4760
Content And Storage
Store at 4°C short term. For long term storage, store at -20°C, avoiding freeze/thaw cycles.
Form
Liquid
Applications
Western Blot
Concentration
0.1 mg/mL
Formulation
PBS with 40% glycerol and 0.02% sodium azide; pH 7.2
Gene Accession No.
Q13562
Gene Symbols
NEUROD1
Immunogen
Recombinant protein corresponding to Human NEUROD1
Quantity
100 μL
Primary or Secondary
Primary
Target Species
Human
Product Type
Antibody
Isotype
IgG
Related Products
Description
- Immunogen sequence: ASFPVHPYSY QSPGLPSPPY GTMDSSHVFH VKPPPHAYSA ALEPFFESPL TDCTSPSFDG PLSPPLSING NFSFKHEPSA EFEKNYAFTM HYPAATLAGA QSHGSIFSGT AAPRCEIPID NIMSFDSHSH HERVMSAQL NeuroD1 is a transcriptional activator that acts as a differentiation factor during neurogenesis
- It has been demonstrated to bind to the insulin gene E-box
- Efficient DNA binding requires dimerization with another basic helix-loop-helix (bHLH) protein
- Defects in NEUROD1 are a cause of maturity onset diabetes of the young type VI (MODY6)
- MODY6 is a form of non-insulin-dependent diabetes mellitus characterized by an autosomal dominant mode of inheritance, onset during young adulthood and a primary defect in insulin secretion.