50-173-6741

PEX5 Mouse anti-Human, Porcine, Rat, Clone: 1C2C3, Proteintech

Manufacturer: Proteintech Group Inc

Select a Size

Pack Size SKU Availability Price
Each of 1 50-173-6741-Each-of-1 In Stock ₹ 44,150.23

50-173-6741 - Each of 1

₹ 44,150.23

In Stock

Quantity

1

Base Price: ₹ 44,150.23

GST (18%): ₹ 7,947.041

Total Price: ₹ 52,097.271

Antigen

PEX5

Classification

Monoclonal

Concentration

1 mg/mL

Formulation

PBS with 50% glycerol and 0.02% sodium azide; pH 7.3

Gene Accession No.

P50542, Q2M2R8

Gene Symbols

Pex5

Immunogen

PEX5 Fusion Protein Ag3268

Quantity

150 μL

Primary or Secondary

Primary

Target Species

Human, Porcine, Rat

Product Type

Antibody

Isotype

IgG1

Applications

Immunohistochemistry (Paraffin), Western Blot

Clone

1C2C3

Conjugate

Unconjugated

Gene

PEX5

Gene Alias

Peroxin 5, Peroxisome receptor 1, PEX5, PTS1 BP, PTS1 receptor, PTS1R, PXR1

Host Species

Mouse

Purification Method

Protein G

Regulatory Status

RUO

Gene ID (Entrez)

100512963, 312703, 5830

Content And Storage

-20°C

Form

Liquid

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Description

  • The product of this gene binds to the C-terminal PTS1-type tripeptide peroxisomal targeting signal (SKL-type) and plays an essential role in peroxisomal protein import
  • Peroxins (PEXs) are proteins that are essential for the assembly of functional peroxisomes
  • The peroxisome biogenesis disorders (PBDs) are a group of genetically heterogeneous autosomal recessive, lethal diseases characterized by multiple defects in peroxisome function
  • The peroxisomal biogenesis disorders are a heterogeneous group with at least 14 complementation groups and with more than 1 phenotype being observed in cases falling into particular complementation groups
  • Although the clinical features of PBD patients vary, cells from all PBD patients exhibit a defect in the import of one or more classes of peroxisomal matrix proteins into the organelle
  • Defects in this gene are a cause of neonatal adrenoleukodystrophy (NALD), a cause of Zellweger syndrome (ZWS) as well as may be a cause of infantile Refsum disease (IRD)
  • Alternatively spliced transcript variants encoding different isoforms have been identified.