62-901-942

CD266 (TWEAK Receptor) Monoclonal Antibody (ITEM-1), Super Bright™ 436, eBioscience™, Invitrogen™

Manufacturer: Invitrogen

Select a Size

Pack Size SKU Availability Price
Each of 1 62-901-942-Each-of-1 In Stock ₹ 32,841.00

62-901-942 - Each of 1

₹ 32,841.00

In Stock

Quantity

1

Base Price: ₹ 32,841.00

GST (18%): ₹ 5,911.38

Total Price: ₹ 38,752.38

Antigen

CD266 (TWEAK Receptor)

Classification

Monoclonal

Concentration

5 μL/Test

Formulation

PBS with BSA and 0.09% sodium azide; pH 7.2

Gene Accession No.

Q9NP84

Gene Symbols

TNFRSF12A

Purification Method

Affinity chromatography

Regulatory Status

RUO

Gene ID (Entrez)

51330

Content And Storage

4° C, store in dark, DO NOT FREEZE!

Form

Liquid

Applications

Flow Cytometry

Clone

ITEM-1

Conjugate

Super Bright 436

Gene

TNFRSF12A

Gene Alias

AI255180; C87282; CD266; FGF-inducible 14; Fgfrp2; fibroblast growth factor regulated protein 2; fibroblast growth factor-inducible immediate-early response protein 14; Fibroblast growth factor-regulated protein 2; FN14; HPIP; TNF receptor superfamily member 12A; TNFRSF12A; Tumor necrosis factor receptor superfamily member 12A; tumor necrosis factor receptor superfamily, member 12A; TweakR; TWEAK-R; Tweak-receptor; type I transmembrane protein Fn14

Host Species

Mouse

Quantity

100 Tests

Primary or Secondary

Primary

Target Species

Human

Product Type

Antibody

Isotype

IgG1 κ

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Description

  • Mouse Monoclonal Antibody TWEAK is a member of the TNF family and was first described as a weak inducer of apoptosis in demonstrate that this relatively small molecule is a fully transformed cell lines
  • Recently, a receptor for TWEAK was isolated by expression cloning from a HUVEC cell cDNA library
  • TweakR is a small protein belonging to TNF receptor super family
  • TweakR is capable of intiating a proliferative signal by binding to TRAF1, 2, and 3
  • TweakR mRNA is upregulated during tissue regeneration in the aorta and liver
  • It may also play a role in vascular cell migration and angiogenesis.